Rifaximin versus lactulose
Review of the literature stimulation of immunity and the normalization of increased mucosal barrier function. The mechanisms behind prebiotics are thought to be the stimulation of the growth of beneficial bacterial species and an increase in colonic short-chain fatty acid concentration. Experimental studies There are not many studies on the attenuation of experimental colitis by probiotics Table V ; . The first study showed a highly protective effect of intracolonic administration of L. reuteri R2LC on acetic acid-induced colitis, which was not simply due to the presence of lactobacilli in general, since another strain, L. reuteri HLC, was ineffective Fabia et al. 1993b ; . The second provided evidence of the antagonistic effect of the continuous intragastric infusion of L. reuteri R2LC on another experimental model, methotrexate-induced enterocolitis, and introduced a new colitis ameliorating strain L. plantarum DSM 9843, also called L. plantarum 299V Mao et al. 1996 ; . Long-term L. reuteri via rectal delivery normalised the levels of increased colonic mucosal-adherent and translocated bacteria and reduced the severity of colitis in IL-10 gene-deficient mice Madsen et al. 1999 ; . Long-term oral administration of a preparation containing bifidobacteria, lactobacilli and Streptococcus salivarius ssp. thermophilus also improved histology, normalised barrier integrity, and down-regulated the inflammatory response in this model Madsen et al. 2001 ; . Histological injury in IL-10 gene-deficient mice has been described as being reduced by oral therapy with L. salivarius ssp. salivarius UCC118 O'Mahony et al. 2001 ; and with L. plantarum 299V Schultz et al. 2002 ; . Oral L. plantarum 299, in contrast, has failed to improve TNBS-induced colitis Kennedy et al. 2000 ; . Studies on the effects of prebiotics on experimental colitis are very scarce. Oral lactulose reduced the levels of increased colonic mucosal adherent and translocated bacteria and attenuated the severity of colitis in IL-10 gene-deficient mice Madsen et al. 1999 ; . This effect seemed to be due to the stimulation of the growth of lactobacilli. Lactulose did not, however, attenuate macroscopic inflammation in severe colitis that had been induced by TNBS, although it reduced systemic endotoxaemia in this model Gardiner et al. 1995 ; . Inulin has been reported to attenuate DSS-induced colitis Videla et al. 2001 ; . The effect of inulin is unlikely to be due to any direct pharmacological action, since topical administration by enema has been ineffective and yet faecal fluid from inulin-fed rats has shown an effect Videla et al. 2001.
Under the hydrogen curves A ; by counting the squares on the linear graph paper and were expressed as parts per million x min x 10 2 after xylose, A was calculated for each of five 1-hr periods, and after lactulose for one 2-hr period 0 to 120 min ; . Total urine, and blood xylose concentrations were measured in duplicate 21 ; . Blood was diluted with 2 vol zinc sulphate 18 g hiter ; + 2 vol barium hydroxide 18 g liter' ; and centrifuged; the supernatant fluid was estimated undiluted. Standard curves, using freshly prepared xylose solutions, were constructed at the beginning and end of every series of determinations. Quantitative jejunal bacterial count 16 ; , and glycochohic acid breath test 22 ; was undertaken in one and two patients, respectively. were determined.
Rifaximin versus lactulose
Suggesting potential adverse clinical liver effects. In contrast, other compounds demonstrate only minor or no signs of hepatotoxicity in the animal species tested, yet cause an increase in serum liver enzymes in more than 10% of humans during early clinical trials reviewed in Fattinger et al., 2001; Pessayre et al., 1985 ; . Our study focused primarily on compounds that were not predicted to result in clinical hepatotoxicity, due to the unresponsiveness of the preclinical species. The common characteristic of the drugs used in this study is the preferential elimination from the body via the biliary pathway, which accounts for at least 50% of total drug clearance. We and others have hypothesized that hepatotoxicity in humans taking these drugs may be associated with drug-mediated inhibition of active canalicular transport of bile components, including, but not limited to, bile acids Kostrubsky et al., 2001; Stieger et al., 2000 ; . These drugs are likely substrates for active liver transporter-mediated uptake and efflux into the bile canaliculi. The transporters that participate in biliary drug elimination also transport endogenous bile components. Therefore, there is a potential for mutual inhibition of drug and bile acid efflux from the liver, resulting in an increase in drug and bile acids retained in the liver over time. We hypothesized that compounds showing greater inhibitory potency for bile acid transport will have a greater risk of being hepatotoxic. To address this issue, we have used an in vitro model of cultured human hepatocytes with an extensive canalicular network. We developed a competition assay between the drug and radioactively labeled bile acid to test whether canalicular efflux of taurocholate can be inhibited in a concentration-dependent manner, and whether this inhibition would correlate with clinical hepatotoxicity. For the current study, we have used six macrolide antibiotics with both high and low incidences of clinical hepatotoxicity. Our data indicate that drugs with greater hepatotoxic risk are stronger inhibitors of taurocholate transport in cultured human hepatocytes. Previously, Fattinger et al. 2001 ; observed an increase in rat serum bile acids after treatment with drugs that cause hepatotoxicity in clinic. These authors found a dose-dependent in.
16 have been most pronounced in the lung where the challenge was concentrated. The early beneficial effects of HRL-3 both on survival and lung injury are consistent with this. In contrast to intravenous E. coli however, intrabronchial challenge ultimately resulted in an established nidus of infection where activated neutrophils would have had an important protective role in the innate immune response. Inhibition of pulmonary neutrophil recruitment by HRL-3 with IB E. coli as was manifested by early reductions in lung lavage and histology neutrophil counts may have resulted in more extensive infection and secondary injury. Later reductions in survival rates and worsened lung injury with HRL3 at 168 h are consistent with this. Although neither blood nor lung bacteria counts were increased in animals receiving HRL-3, this may be because administration of antibiotics 2 h before laboratory measures in the present study altered the results of microbiology testing. It is also possible that the microbiology techniques employed were insensitive to the effects HRL-3 may have had on bacterial clearance. Consistent with a potentially adverse effect of HRL-3 on host defense in the present study however, in a recently published phase 2 clinical trial in trauma patients, another L-selectin directed monoclonal antibody was found to be associated with dose related increases in the rate of infection 40 ; . Alternatively however, the detrimental effects of HRL-3 on survival rates or lung injury during intrabronchial infection in the present study may have been because it either interfered with a later adaptive immune response necessary for the clearance of an intrabronchial nidus of infection or with subsequent reparative processes. The present findings with HRL-3 are consistent with prior studies performed in this laboratory in a large animal model, in which monoclonal antibodies directed at leukocyte integrin adhesion molecules improved survival with intravenous tumor.
Duphalac lactulose indication
Shown to be relevant in the etiology of low back, abdominal, and pelvic pain syndromes 20-23 ; . At first glance, this retrospective study appears to confirm the immediate and longterm effectiveness of TrP injections in abdominal trigger points. A reported 89 percent and 77 percent experienced immediate or long-term relief respectively. However, the true effects of the injections are difficult to interpret. The authors did not report how they defined TrPs or TrP injections. What criteria did they use to palpate abdominal TrPs? Did they determine the presence of a taut band and the most tender region within that taut band as suggested by Simons, Travell and Simons 7 ; ? The authors do not mention whether they attempted to elicit local twitch responses or whether they infiltrated the general vicinity of a point of tenderness. Intramuscular TrP injections are only effective if the needle elicits local twitch responses 24 ; . The number of follow-up injections and the time line of follow-up appointments varied greatly. With such a wide range, retrospective chart reviews do not provide sufficient assurances that reported long-term relief is primarily due to any particular intervention. The authors expressed surprise about the lack of effectiveness of adding a steroid to local anesthetics. This may indicate a lack of familiarity with the integrated trigger point hypothesis 7 ; . Steroids are most effective when treating inflammatory conditions and there is no current evidence that TrPs are associated with local inflammation. The use of steroids is not recommended for the injection of TrPs and may actually lead to destruction of muscle fibers. There is also no evidence that using long-acting anesthetics are more effective than short acting anesthetics in the treatment of TrPs. Long-acting anesthetics may actually be more myotoxic. The authors reported using a one percent to two ercent lidocaine solution. A recent study indicated that using a 0.25 ercent lidocaine solution is more effective 25 ; , while reducing its myotoxicity. The observation that subjects with a surgical scar required significantly more TrP injections is consistent with Lewit and Olsanska's notion that scars are a common source of myofascial pain 26 ; . Prospective studies on the effectiveness of treat.
TO THE EDITOR: Pramipexole, a dopamine receptor agonist, is currently indicated for treatment of idiopathic Parkinson's disease. There is also preliminary evidence suggesting that it may have a role in the treatment of bipolar depression 13 ; . Pramipexole is generally well tolerated, but there are occasional reports of induction of hypomania. Singh et al. 4 ; described a case of mania in a patient with Parkinson's disease with no personal or family history of bipolar disorder. We report the case of a woman with bipolar II disorder who developed a manic episode following treatment of depression with pramipexole. Written informed consent was obtained from the patient after the procedure had been fully explained and lantus.
Lactulose for women
History of schizophrenia on treatment with lithium, perphenazine, and trihexyphenidil long-term. She took traditionally boiled Lingzhi slices for several years without any untoward effect. She changed to taking Lingzhi powder, one capsule containing 400 mg of Lingzhi extract per day, for two months before having the episode of jaundice and coma due to fulminant hepatitis. Her previous liver biochemical profiles were within normal limits. On admission, the biochemical profiles included albumin 30.4 g l normal 43.1-53.3 ; , total bilirubin 690.3 mmoles l normal 0-17.1 ; , direct bilirubin 504.9 mmoles l normal 0-5 ; , alkaline phosphatase 132 U l normal 50-136 ; , gamma glutamyltranspeptidase 59 U l normal 5-55 ; , aspartate aminotransferase 280 U l normal 15-37 ; , alanine aminotransferase 276 U l normal 30-65 ; and prothrombin time 55.3 seconds normal 10-13 ; , international normalized ratio INR ; 4.66 normal 0.85-1.10 ; . The serological tests for anti HAV IgM, anti-HBcIgM and anti-HCV were negative. Acute hepatic failure was considered. N-acetyl cysteine 100 mg kg day with lactulose was given. Endotracheal intubation with mechanical ventilator and gastric intubation were performed. Antibiotic prophylaxis with third generation cephalosporin Cefotaxime ; was prescribed. Fresh frozen plasma and.
Lactulose
Anex convened the 20 Years of NSP Success Planning Committee to organise the first in a series of events to mark the 20th Anniversary of Australian NSP. Attendees heard from a range of speakers who were instrumental in the establishment of NSP in Australia and lavender.
The model permits estimation of the number of cardiovascular deaths in each category by assuming case-fatality rates of 30% for MI and HS, 25% for IS, and 2% for VTE. With these case-fatality rates, the number of deaths corresponding to each of the previous categories at different ages are shown in Figure 1 [panels v ; to viii ; ], with the corresponding OC-attributable mortality in Table V. The.
Authors' reply Editor--We agree with Battle et al that the use of probiotics remains but one method of manipulating faecal flora as a means of controlling antibiotic induced diarrhoea. Using lactulose is an interesting idea, although altering bacterial flora by introducing an organism that can colonise the bowel as well as modify the proliferation of other organisms seems easier. Probiotics also have an antiendotoxin effect with an added benefit of stimulating an immunoglobulin response in the gut wall.1 Furthermore, the use of lactulose is limited by lack of knowledge on the optimum dose at which it can have an effect on gut flora while not causing diarrhoea. We agree that Clostridium difficile accounts for only 10-25% of cases of antibiotic associated diarrhoea. It accounts, however, for the most cases of colitis that occur and remains treatable once diagnosed.2 Our recommendations on the use of probiotics were not specific to elderly people--we did point out that a trial was needed in this age group. The supposed error in table 1 relates to the percentage free of diarrhoea in the active and placebo groups in the study by Gotz et al.3 Our analysis was performed by using the numbers as mentioned 8.3% in the active group v 21% in the placebo group the table highlighted the numbers in the groups when other treatments likely to induce diarrhoea were excluded. The study by Borgia et al was excluded because it primarily studied effects on body weight.4 The study by Colombel et al did not provide enough information on the number of cases of diarrhoea alone diarrhoea was grouped as a side effect along with nausea, vomiting and abdominal pain ; . 5 We are criticised for these exclusions and yet for including studies analysed on a "per protocol" basis, an acceptable scientifically rigorous procedure. We acknowledge the risks posed by probiotics, but most complications occurred in immunosuppressed individuals and people with other life threatening illnesses. This was mentioned in our paper, and we would leave it to clinicians to decide the individual suitability of patients to receive probiotics. Our proposals about these agents seek not to replace antibiotic and infection control policies but to supplement them. A and lenalidomide.
Lactulose dosing instructions
| Lactulose for children's constipationPrecautions: enulose lactulose may not produce any result for between 24 and 48 hours.
Adjacent to the great room is a white tile-floored sunroom where crisp white bead board covers most of the walls. Albright painted the blue sky and fluffy white clouds that adorn the ceiling. Another Claire Joyce glitter painting, starring a trio of cavorting bunnies, hangs above a large formal dining table. The formerly dark wood table, a handme-down from Albright's grandmother, now sports clean white paint and is surrounded by industrial brushed stainless steel chairs in a perfect marriage of nostalgia and contemporary sleekness. In spite of its overall tone of calm, cool sophistication, the vibrant character in every corner of this home aptly conveys the warm personality and casual attitude of its owners. From academic obsessions to family nostalgia to their beloved animals, this fun-loving couple has created a comfortable space that is both practical and elegant. Melissa Link is a freelance writer living in Athens and leuprolide.
Introduction Epidemiological studies continue to support the premise that dietary intake of Allium vegetables, especially garlic, may offer protection against the risk of different types of malignancies including cancer of the prostate 1-4 ; . For example, Hsing et al. 4 ; examined the association between Allium vegetable intake and prostate cancer risk in a population-based study involving 238 histologically confirmed cases and 471 controls. The risk of prostate cancer was found to be significantly lower in men consuming 10g day of total Allium vegetables than in men with total Allium vegetable intake of 2.2 g day 4 ; . Laboratory studies indicate that anticarcinogenic effect of Allium vegetables is due to organosulfur compounds OSCs ; that are generated upon processing cutting or chewing ; of these vegetables 5, 6 ; . Garlic-derived OSCs including diallyl sulfide, diallyl disulfide DADS ; and diallyl trisulfide DATS ; have been shown to offer significant protection against cancer in animal models induced by a variety of chemical carcinogens 7-13 ; . For example, cancer prevention by naturally occurring OSC analogues has been observed against dimethylhydrazine-induced colon cancer in rats, benzo[a]pyrene-induced forestomach and pulmonary carcinogenesis in mice, azoxymethane-induced colon cancer in rats, and N-methyl-N-nitrosourea and 5-b]pyridine-induced mammary cancer in rats 7-13 ; . Prevention of chemically-induced cancers in animal models by OSCs is believed to be due to their ability to increase detoxification of activated carcinogenic metabolites through induction of Phase II enzymes such as glutathione transferases and or inhibit carcinogen activation through inhibition of Phase I enzymes 14-17 ; . In addition, oral administration of DADS significantly suppresses growth of H-ras oncogene transformed NIH 3T3 tumor xenografts in athymic mice by inhibiting membrane association of p21H-ras 18, 19.
Canadian Lactulose
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Accepted in Principle. Added clarification that the translation is done here by the FC-1 translation block as follows: "10GFC does not support ARB or NOS. ARB must be mapped by the FC-1 translation block to the XGMII Idle character since XGXS devices only delete Primitive Sequences or the 10GFC Idle characters. NOS does not appear on XGMII or XAUI and is mapped by the FC-1 translation block to Remote Fault RF and levalbuterol.
Additional copies More detailed information is available in the New Brunswick Student Drug Use 2002: Technical Report. To obtain a copy of the Technical Report or for additional copies of the Highlights Report, contact: New Brunswick: Dr. Jian Liu Provincial Epidemiology Service Department of Health and Wellness PO Box 5100, Fredericton, NB E3B 5G8 Telephone: 506-453-3092 Fax : 506-453-2780 Prince Edward Island: Linda Van Til Department of Health and Social Services PO Box 2000, Charlottetown, PEI C1A 7N8 Nova Scotia: Brian Wilur Drug Dependency Services Operations and Regional Support Branch Department of Health PO Box 488, Halifax, NS B3J 2R8.
Over the last years most of the legislative initiatives taken by the Directorate-General for Energy and Transport of the European Commission have created great concerns. Resulting in strikes all over Europe, in several sectors of the maritime industry such as stevedores, and combined with a warm welcome from certain Members States such as Austria, happy to see how the European institutions fight against the chaotic Alpine road network. By submmitting this 14 measures programm it appears clear that Ms. Loyola de Palacio and her team in Brussels, are determined to do something about the actual inconvenient European transport network, and to do it fast. Her perseverance was not heard in her intention to shorten the deadline of incorporating the legislation of single hull oil carriers, and the Prestige incident came along. Should the Member States have paid more consideration to Ms. De Palacio concerns, the Prestige would not have been aloud to sail across European waters by the time of the accident and levamisole.
Polyethylene glycol and lactulose are prescription laxatives that belong to the category of osmotic laxatives, which are poorly absorbed or nonabsorbed substances that increase the secretion of water into the intestine. Other osmotic laxatives, which are available OTC, thus they are not discussed in detail in this article, include salts eg, magnesium and phosphate ; and sugar alcohols eg, sorbitol or mannitol ; . Results from 6 randomized, double-blind, placebo-controlled trials, 2 randomized comparative trials, and other studies evaluating lactulose and PEG prompted an American College of Gastroenterology Task Force to give both agents a Grade A recommendation ie, a recommendation supported by 2 or more Level 1 trials without conflicting evidence from other Level 1 trials ; .1 Two of the studies comparing lactulose with placebo and 3 of the studies comparing PEG with placebo found that active treatment improved stool consistency and increased the number of bowel movements per day, 1 whereas 2 openlabel studies comparing PEG to lactulose found that PEG resulted in greater stool frequency, less straining, and better overall effectiveness.2, 3 A secondary analysis of 1 of these studies2 suggested that PEG was also more cost effective than lactulose.4 In the other study, which compared both agents in elderly patients for 6 months, weekly stool frequency was significantly higher and the incidence of hard stools significantly lower in those patients receiving PEG.3 In both of these trials, patients Table. Effects of Tegaserod Versus Placebo on Chronic Constipation taking PEG reported fewer side effects, particularly bloating, and were more satisfied with therapy than patients Tegasrod 6 mg twice daily Placebo taking lactulose and lactulose.
Lactulose breath test preparation
Under section 6 of the Mental Health Law, either the Director of MHRH or the Director of the Department of Corrections may file a petition with the Court to transfer an inmate who is awaiting trial or incarcerated for conviction of a crime from the ACI to the Forensic Unit. Id. 40.1-5.3-6. Notice and levemir.
XAFFIRE, INC. DELAWARE CORPORATION ; SUITE 200 100 SUPERIOR PLAZA WAY SUPERIOR, CO 80027 FOR: DOWNLOADABLE COMPUTER PROGRAMS FOR MONITORING AND MANAGING DISTRIBUTED COMPUTER SOFTWARE APPLICATIONS AND THEIR ASSOCIATED NETWORKS.
In IL-6 and IL-12 were identified in the burn injury model, whereas changes in TNF- , IL-1 , IL-12, IL-6, IL-10, IFN- , and GM-CSF were increased in the endotoxemia model unpublished data ; . Although MIF mediates a late cardiac dysfunction in each of these models, the underlying pathogenesis is considerably different. Moreover, these findings suggest that burn injury-induced changes in MIF are not solely due to LPS. The cytokine MIF plays a significant role in the late cardiac dysfunction associated with burn injury. Although we have previously identified that MIF itself has cardiodepressive effects ex vivo 26 ; , it is not clear whether this direct effect accounts for any of the observed cardiac dysfunction after burn injury. MIF may indirectly induce cardiac dysfunction by stimulating the release of downstream cytokines and or act synergistically with other cytokines i.e., IL-6 ; to mediate late burn injury-associated cardiac dysfunction. Because MIF release is delayed after burn injury, the development of small molecule inhibitors i.e., tautomerase inhibitors ; that appear to have in vitro anti-MIF activity 18 ; may make MIF a good potential therapeutic target and levetiracetam.
1. Minekus M, Smeets-Peeters M, Bernalier A, Marol-Bonnin S, Havenaar R, Marteau P, Alric M, Fonty G, Huis in 't Veld JHJ. A computer-controlled system to simulate conditions of the large intestine with peristaltic mixing, water absorption and absorption of fermentation products. Appl Microbiol Biotechnol 1999; 53: 108-14. Venema K, van Nuenen HMC, Smeets-Peeters M, Minekus M, Havenaar R. TNO's in vitro large intestinal model: an excellent screening tool for functional food and pharmaceutical research. Ernhrung Nutrition 2000; 24 12 ; : 558-64. Venema K, van Nuenen HMC, van den Heuvel EG, Pool W, van der Vossen JMBM. The effect of lactulose on the composition of the intestinal microbiota and short-chain fatty acid production in human volunteers and a computer-controlled model of the proximal large intestine. Microb Ecol Health Disease 2003; 15: 94-105. Van Nuenen HMC, Meyer PD, Venema K. The effect of various inulins and Clostridium difficile on the metabolic activity of the human colonic microbiota in vitro. Microb Ecol Health Disease 2003; 15: 137-44. Gibson GR, Roberfroid MB. Dietary modulation of the human colonic microbiota: introducing the concept of prebiotics. J Nutr 1995; 125: 1401-12. Cummings JH, Roberfroid MB, Andersson H, Barth C, Ferro-Luzzi A, Ghoos Y, Gibney M, Hermonsen K, James WPT, Korver O. A new look at dietary carbohydrate: chemistry, physiology and health. Eur J Clin Nutr 1997; 51: 417-23. Jorgensen J, Mortensen PB. Hydrogen sulfide and colonic epithelial metabolism - Implications for Ulcerative colitis. Dig Dis Sci 2001; 46 8 ; : 1722-32. Chadwick VS, Anderson RP. Microorganism and their products in inflammatory bowel disease. In: MacDermott RP, editor. Inflammatory bowel disease. New York: Elsevier, 1992: 241-58. Cummings JH, MacFarlane GT. The control and consequences of bacterial fermentation in the human colon. J Appl Bacteriol 1991; 70: 443-59. Gibson GR, MacFarlane GT. Human colonic bacteria: role in nutrition, physiology, and pathology. Florida: CRC press, 1995. Smith EA, MacFarlane GT. Formation of phenolic and indolic compounds by anaerobic bacteria in the humen large intestine. Microb Ecol 1997; 33: 180-8. Ruseler-van Embden JGH, Schouten WR, van Lieshout LMC. Pouchitis: result of microbial imbalance? Gut 1994; 35: 658-64. Cummings JH, Englyst HN. Fermentation in the large intestine and the available substrates. J Clin Nutr 1987; 45: 1243-55. Cummings JH, Gibson GR, MacFarlane GT. Quantitative estimates of fermentation in the hind gut of man. Acta Vet Scan Suppl 1989; 86: 76-82. Foitzik T, Kruschewski M, Kroesen A, Buhr HJ. Does microcirculation play a role in the pathogenesis of inflammatory bowel diseases. Int J Colorectal Dis 1999; 14: 29-34. Eckmann L, Kagnoff MF, Fierer J. Intestinal epithelial cells as watchdogs for the natural immune system. Trends Microbiol 1995; 3: 118-20. Wachtershauser A, Stein J. Rationale for the luminal provision of butyrate in intestinal diseases. Eur J Nutr 2000; 39 4 ; : 164-71 and lantus.
Lactulose dosage dose
Lactone and furosemide] ; , encephalopathy lactulose ; , and cirrhosis ursodeoxycholic acid ; . Other comedications were primarily for diabetes, pain, and nutritional supplementation. Pharmacokinetics. The values of the telbivudine single-dose pharmacokinetic parameters in subjects with mild, moderate, and severe hepatic impairment were comparable to those in subjects with normal hepatic function Table 2 ; . Plasma concentration-time profiles were similar in subjects with normal and impaired hepatic function, as shown in Fig. 1. Telbivudine concentrations rapidly increased to a mean Cmax of 2.8 g ml median, 2.5 g ml ; at median Tmax of 2.5 h postdosing in subjects with normal hepatic function. In subjects with hepatic impairment, the mean Cmax of telbivudine ranged from 3.4 to 3.7 g ml median range, 2.8 to 3.2 g ml ; , and the median and levonorgestrel.
1. Knapp Jr FF. Kropp J. Iodine-123-labeled fatty acids for myocardial SPECT: current status and future perspectives. Ear J Nuc-Med 1995: 22: 361-381. Knapp Jr FF. Kropp J. Goodman MM. et a!. The development of iodine-123-methylbranchcd fatty acids and their applications in nuclear cardiology. Ann Nuc-Med 1993: 7: 1-14. Knapp FF Jr. Goodman MM. Ambrose KR. et al. The development of radioiodinated 3-mcthyl-branched fatty acids for evaluation of myocardial disease by single photon techniques. In: van der Wall EE. ed. NonlKVQStveimaging of cardiac melaholiatn. Dordrecht. The Netherlands: Martius Nijhoff Publishers; 1987: 159-201. 4. Fujibayashi. Y. Yonekura Y. Kawai K. et al. Basic studies on acid BMIPP ; for myocardial functional diagnosis: effect of beta-oxidation inhibitor. Jpn J Nuc- ed 1988: 25: 1131 -1135. M 5. Kropp J. Kohler U. Knapp Jr FF. Biersack HJ. 15- p-[I-123]iodophenyl ; -3-R, S-methylpentadccanoic acid to evaluate ischemia in patients with coronary artery disease [Abstract]. Eur J Nuc- ed 199l; 18 suppl ; : 650. M 6. Kropp J. Juergans M. Glaenzer K. et al. Evaluation of ischemia and myocardial viability in patients with coronary artery disease CAD ; with iodine-123-labeled l5- p-iodophenyl ; -3-R. S-methylpentadecanoic acid BMIPP ; . Ann Nuc- ed 1993: M 7: 93-100. 7. De Geetcr F. Franken PR. Knapp Jr FF. Bossuyt A. Relationship between blood flow and fatty acid metabolism in subacute myocardial infarction: a study by means of technetium-99m-sestamibi and iodine-123-bcta-mcthyl iodophenyl-pentadecanoic acid. Eur J Nuc- ed 1994: 21: 283-291. M 8. Franken PR. De Geeter F. Dcndale P. et al. Abnormal free fatty acid uptake in subacute myocardial infarction after coronary thrombolysis: correlation with wall motion and inotropic reserve. J Nuc- ed 1994: 35: 1758-1765. M 9. Tamaki N. Kawamoto M, Yonekura Y. et al. Regional metabolic abnormality in relation lo perfusion and wall motion in patients with myocardial infarction: assess ment with emission tomography using iodinated branched fatty acid analogue. J NucMed 1992: 33: 659-667. Tamaki N. Kawamoto M. Yonekura Y. el al. Assessment of fatty acid metabolism.
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Taking lactulose in pregnancy
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